PectaSol Modified Citrus Pectin Powder Super-Nutrient to Support Cellular & Immune Health, Joint Support - 454 Grams - Formulated by Dr. Isaac Eliaz of ecoNugenics

Product Information

If you’re concerned about possible sensitivities to MCP, consult with your practitioner before use, and start with a lower dose to see how your body responds.

Modified Citrus Pectin: Your Guide to Benefits, Risks, and Usage

We offer three ways to take PectaSol, all offer the same benefits and the form you take is all based on personal preference! Finally, several studies have shown that orally taken pectin decreases the risk of intestinal infection and of diarrhea in children by favoring the growth of “good” bacteria in the colon (e. In conclusion, MCP displays many anti-metastatic properties demonstrated both in vitro and in vivo, in various malignancies. Inhibition of spontaneous metastasis in a rat prostate cancer model by oral administration of modified citrus pectin. This allows PectaSol-C® to absorb into the circulation and deliver total-body benefits related to cellular health, immunity and more.GCS-100, which is a commercially available form of modified pectin, has been shown to be efficient against different lines of multiple myelomas some of them resistant to chemotherapy, by inducing caspase-3 and -8 activation as well as PARP cleavage.

PectaSol-C (Modified Citrus Pectin) Lime Infusion, 551g

Furthermore, HG-rich pectin with a high DE competes with LPS for TLR4 binding, hence preventing inflammatory cell activation. Powerful synergistic potential: A number of studies highlight the ability of this MCP to enhance the efficacy and mechanisms of other therapies. Galectin-3 Deficiency Ameliorates Fibrosis and Remodeling in Dilated Cardiomyopathy Mice with Enhanced Mst1 Signaling,” Am.Thousands of studies identify galectin-3 as an upstream target for our most critical areas of health including cytokine cascades, immune response, cellular regulation, cardiovascular and kidney function, and others. Colon carcinogenesis is a multi-step process that results from disruption of the balance between proliferation of colonocytes at the base of the crypt and loss of colonocytes at the luminal surface due to apoptosis.